Article
Suppression of Bilirubin Production in the Crigler-Najjar Type I Syndrome: Studies with the Heme Oxygenase Inhibitor Tin-Mesoporphyrin
Richard A. Galbraith, George S. Drummond and Attallah Kappas
Pediatrics February 1992, 89 (2) 175-182;
Richard A. Galbraith
George S. Drummond

Submit a Response to This Article
Jump to comment:
No eLetters have been published for this article.
In this issue
Log in
You will be redirected to aap.org to login or to create your account.
Suppression of Bilirubin Production in the Crigler-Najjar Type I Syndrome: Studies with the Heme Oxygenase Inhibitor Tin-Mesoporphyrin
Richard A. Galbraith, George S. Drummond, Attallah Kappas
Pediatrics Feb 1992, 89 (2) 175-182;
Jump to section
Related Articles
- No related articles found.
Cited By...
- A Nanoceutical Agent for Chemoprevention of Bilirubin Encephalopathy
- Repurposing Tin Mesoporphyrin as an Immune Checkpoint Inhibitor Shows Therapeutic Efficacy in Preclinical Models of Cancer
- Pharmacological and Clinical Aspects of Heme Oxygenase
- Pharmacology Review: Tin Mesoporphyrin for the Prevention of Severe Neonatal Hyperbilirubinemia
- A Method for Interdicting the Development of Severe Jaundice in Newborns by Inhibiting the Production of Bilirubin
- Regulation of human heme oxygenase in endothelial cells by using sense and antisense retroviral constructs
- Zinc Protoporphyrin: A Metabolite with a Mission
- Control of Severe Hyperbilirubinemia in Full-term Newborns With the Inhibitor of Bilirubin Production Sn-Mesoporphyrin
- Lack of Deafness in Crigler-Najjar Syndrome Type 1: A Patient Survey
- Zinc porphyrins: Potent inhibitors of hematopoieses in animal and human bone marrow