PEDIATRICS Vol. 97 No. 1 January 1996, pp. 151-152
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Early Prediction of the Development of Microcephaly After Hypoxic-Ischemic Encephalopathy in the Full-term Newborn

Nadia Badawi MBBCh, MSc, DCH, MRCP(I)1, Jennifer J. Kurinczuk MBChB, MSc, MFPHM, FAFPHM1, Eve Blair PhD1, John Keogh MBBS, MRCOG, MRACOG2, and Fiona Stanley MD, MSc, FFPHM, FAFPHM, MFCCH1

1 Institute for Child Health Research, Department of Paediatrics, University of Western, Australia
2 King Edward Memorial Hospital for Women, Department of Paediatrics, University of Western, Australia

We read with interest the article by Cordes et al.1 This article raises an important issue concerning assumptions made with respect to the significance of intrapartum and neonatal observations. The population of infants studied was 54 full-term newborns born after uneventful pregnancies and diagnosed as having acute hypoxic-ischemic encephalopathy (HIE) that was intrapartum in origin. The underlying assumption of this diagnosis was that if there is no obvious antenatal cause of abnormal neonatal neurological signs, then any suboptimal intrapartum observation identifies the pathology as being intrapartum in origin.