PEDIATRICS Vol. 96 No. 2 August 1995, pp. 383
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ABOLITION OF ANAPHYLAXIS BY TARGETED DISRUPTION OF THE HIGH-AFFINITY IMMUNOGLOBULIN E (IgE) RECEPTOR agr CHAIN

Kathleen May MD1 and Andrew Liu MD1

1 Denver, CO

Absence of the high-affinity IgE receptor in mice does not alter mast cell development or numbers, but completely protects these mice from IgE-mediated anaphylaxis. Therefore, it can be surmised that the high-affinity IgE receptor is necessary for allergic anaphylaxis (inmice).