PEDIATRICS Vol. 66 No. 6 December 1980, pp. 967-971
This Article
Right arrow Full Text (PDF)
Right arrow P3Rs: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when P3Rs are posted
Right arrow Alert me if a correction is posted
Services
Right arrow E-mail this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My File Cabinet
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via CrossRef
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Percy, A. K.
Right arrow Articles by Locke, G. E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Percy, A. K.
Right arrow Articles by Locke, G. E.

Cerebral Cysticercosis

Alan K. Percy MD1, Sharon E. Byrd MD1, and George E. Locke MD1

1 Department of Pediatrics, Radiology, and Surgery, Charles R. Drew Postgraduate Medical School, and Martin Luther King, Jr, General Hospital, Los Angeles

In this country, cerebral cysticercosis is not commonly implicated as the etiology of an otherwise uncomplicated seizure disorder occurring during childhood. Nine children with neurologic symptomatology and radiographic (computed tomography) evidence consistent with cerebral cysticercosis have been evaluated. Each patient was born and resided for some period (1frac12 to 10 years) in an endemic area. Principal symptomatology consisted of a generalized seizure disorder. EEGs were normal in five patients and nonspecific in two. Serologic studies on serum and CSF were negative in 8/9 patients including the single patient requiring surgical intervention for excision of a cysticercal cyst. Cerebral nuclide scanning was not helpful and skull radiography was used to diagnose intracerebral calcifications in six patients. Computed tomography revealed multiple calcifications in six patients and a solitary lesion in one. Five patients had enhancing cystic lesions including three of those with diffuse calcifications. Since the natural history of cerebral cysticercosis presenting in this fashion is benign, symptomatic treatment is often sufficient. Anticonvulsants generally provide adequate seizure control. In one patient, cyst removal was necessary to achieve seizure control. On the basis of this experience, conclusions are (1) that the incidence of cerebral cysticercosis in exposed children is higher than commonly appreciated, and (2) that computed tomography may assist in the establishment of the diagnosis.

Submitted on February 27, 1980
Accepted on April 14, 1980




This article has been cited by other articles:


Home page
J Child NeurolHome page
P. Singhi, M. Ray, S. Singhi, and N. Khandelwal
Clinical Spectrum of 500 Children With Neurocysticercosis and Response to Albendazole Therapy
J Child Neurol, April 1, 2000; 15(4): 207 - 213.
[Abstract] [PDF]