PEDIATRICS Vol. 65 No. 2 February 1980, pp. 294-297
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Ammonia Encephalopathy Secondary to Urinary Tract Infection with Proteus mirabilis

Bernard Samtoy MD1 and Martin M. DeBeukelaer MD1

1 Department of Pediatrics, Medical University of South Carolina, Charleston

Hyperammonemia with coma, tachypnea, and respiratory alkalosis developed in a 3-year-old boy with prune-belly syndrome during a urinary tract infection with Proteus mirabilis. Hyperammonemia is thought to have resulted from the production within the massively dilated urinary tract of excessive amounts of ammonia due to bacterial urease, and its subsequent reabsorption into the systemic circulation. The patient rapidly improved following parenteral antibiotic therapy and continuous catheter drainage of the urinary tract.

Submitted on November 6, 1978
Accepted on May 14, 1979




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