PEDIATRICS Vol. 27 No. 3 March 1961, pp. 354-361
This Article
Right arrow Full Text (PDF)
Right arrow P3Rs: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when P3Rs are posted
Right arrow Alert me if a correction is posted
Services
Right arrow E-mail this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My File Cabinet
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via CrossRef
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Kaplan, E.
Right arrow Articles by Hsu, K. S.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Kaplan, E.
Right arrow Articles by Hsu, K. S.

DETERMINATION OF ERYTHROCYTE SURVIVAL IN NEWBORN INFANTS BY MEANS OF CR51-LABELLED ERYTHROCYTES

Eugene Kaplan M.D.1 and Ku Shin Hsu M.D.1

1 Departments of Pediatrics of the Sinai Hospital of Baltimore, Inc., and the Johns Hopkins University School of Medicine

There is no difference in the rate of Cr51 elution from intact cord or adult erythrocytes Variable results of survival studies suggest that placental blood is unsuited for valid and precise measurement of erythrocyte life span. Decreased survival is not present for the erythrocytes of the normal full-term newborn infant in the first 5 days of life when compared with the adult. In the premature infant of the same age shortened life span is found of erythrocytes produced in utero. This may contribute in some degree to the characteristic increase in severity of premature anemia and hyperbilirbinemia. The apparent reduction in erythrocyte life span during the second and third months of life in both premature and full-term infants ma reflect an older erythrocyte population resultant from decreased erythropoiesis in the first weeks of life.




This article has been cited by other articles:


Home page
 Arch. Dis. Child. Fetal Neonatal Ed.Home page
A S Dhillon, P J Darbyshire, M D Williams, and J G Bissenden
Massive acute haemolysis in neonates with glucose-6-phosphate dehydrogenase deficiency
Arch. Dis. Child. Fetal Neonatal Ed., November 1, 2003; 88(6): F534 - 536.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Physiol. Regul. Integr. Comp. Physiol.Home page
R. A. Brace, C. Langendorfer, T.-B. Song, and D. M. Mock
Red blood cell life span in the ovine fetus
Am J Physiol Regulatory Integrative Comp Physiol, October 1, 2000; 279(4): R1196 - R1204.
[Abstract] [Full Text] [PDF]

Home page
 CLIN PEDIATRHome page
W. R. Metge, W. T. Foulk, and J. C. Dower
Physiologic Mechanisms of Jaundice in the Newborn: A Survey of New Insights
Clinical Pediatrics, October 1, 1962; 1(1): 152 - 156.
[PDF]