Conflicting opinions have been expressed as to the mechanism of pulmonary and peripheral congestion frequently observed in the oliguric stage of acute glomerulonephritis. The factors that have received major consideration as responsible for the circulatory complications are hypertension, myocardial damage, and hypervolemia.
Hemodynamic studies utilizing the technic of cardiac catherterization were carried out in 7 hypertensive and edematous patients in the early phase of acute glomerulonephritis to secure information regarding circulatory changes in this disease.