POLICY STATEMENT |
Fatal lead encephalopathy has disappeared and blood lead concentrations have decreased in US children, but approximately 25% still live in housing with deteriorated lead-based paint and are at risk of lead exposure with resulting cognitive impairment and other sequelae. Evidence continues to accrue that commonly encountered blood lead concentrations, even those less than 10 µg/dL, may impair cognition, and there is no threshold yet identified for this effect. Most US children are at sufficient risk that they should have their blood lead concentration measured at least once. There is now evidence-based guidance available for managing children with increased lead exposure. Housing stabilization and repair can interrupt exposure in most cases. The focus in childhood lead-poisoning policy, however, should shift from case identification and management to primary prevention, with a goal of safe housing for all children.
Key Words: child lead environmental exposure chelation therapy succimer cognition clinical trials housing prevention behavior
Abbreviations: CDC, Centers for Disease Control and Prevention AAP, American Academy of Pediatrics EPA, Environmental Protection Agency CNS, central nervous system EP, erythrocyte protoporphyrin EDTA, ethylenediaminetetraacetic acid TLC, Treatment of Lead-Exposed Children HUD, Department of Housing and Urban Development
The following policy statement has been revised:
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