PEDIATRICS Vol. 108 No. 3 September 2001, p. e50
Received Dec 27, 2000; accepted May 10, 2001.
,
,
,
, 
From the * Center for Cardiovascular Genetics, Department of
Medicine, Royal Free and University College London Medical School,
London, United Kingdom; and Departments of Objectives. To examine the
genotype:phenotype association in children compared with their
parents.
Methods. Variations at 4 key gene loci, namely
lipoprotein lipase (LPL S447X), hepatic lipase
(HL Results. The frequencies of the rare alleles of the
HL Conclusions. All genotypes were associated with clear
relationships to plasma lipid levels in adults, but the effects were
weaker in their children, unless stressed by body fat.
atherosclerosis, cardiovascular disease, child, lipids, genetics.
Medicine and § Pediatrics
and
Division of Epidemiology, Joseph Mailman School of Public
Health, Columbia University, New York, New York.
480C>T), cholesteryl ester transfer protein
(CETP TaqIB), and apolipoprotein CIII
(APOC3
455T>C and
482C>T), were examined in
children (n = 495) and their parents (n = 353) in the Columbia University BioMarkers
Study, 1994 to 1998.
480C>T and APOC3
455T>C and
482C>T (but not LPL S447X or CETP
TaqIB) were significantly lower in non-Hispanic white
participants compared with Hispanics. Overall, genotype effects seen in
the adults were weaker in the children, although similar trends were
seen. In an examination of the effect of body fat on the genotypic
effects in the children, there was significant HL
480C>T:sum of skinfold interaction.